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Improving the efficacy and safety in the treatment of resistant forms of chronic myelogenous leukemia, including the variant with T315I mutation. Efficacy against clones of CML tumor cells with T315I mutation in the proto-oncogene BCR-ABL. Suppression of the emergence of resistance. Suppression of the emergence of resistant forms of CML in experiments with directed mutagenesis of CML tumor cells. When resistant to two or more Abl inhibitors.